{"id":7793,"date":"2017-07-14T15:03:31","date_gmt":"2017-07-14T07:03:31","guid":{"rendered":"http:\/\/tstcc.lgznkxsngp-eqg35k5y13xn.p.temp-site.link\/?page_id=7793"},"modified":"2026-09-25T14:52:58","modified_gmt":"2026-09-25T06:52:58","slug":"%e8%84%8a%e6%9f%b1%e5%81%b4%e5%bd%8e","status":"publish","type":"page","link":"https:\/\/tstcc.hk\/en\/conditions-symptoms\/%e8%84%8a%e6%9f%b1%e5%81%b4%e5%bd%8e\/","title":{"rendered":"Scoliosis"},"content":{"rendered":"

Chiropractic Care for Scoliosis<\/h3>\n
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Classifications, Symptoms, and Advanced Neurotransmitter Research (Tsim Sha Tsui)<\/strong><\/div>\n
Scoliosis (derived from the Greek word meaning \"crooked\" or \"curved\") is a complex medical condition characterized by an abnormal, three-dimensional curvature and rotation of the spine<\/strong>. This curvature can lean to the left or right, presenting in the thoracic spine (upper\/mid back), the lumbar spine (lower back), or spanning across both as a thoracolumbar curve. In many clinical presentations, the spine develops a secondary compensatory curve, appearing as an \"S-shape\" on standing digital X-rays.<\/div>\n
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Located in the heart of Tsim Sha Tsui, Kowloon<\/strong>, Your Spine Centre provides specialized, non-surgical, and drug-free scoliosis screenings and functional rehabilitation under the management of registered chiropractors.<\/div>\n
Contact our Tsim Sha Tsui chiropractic team: [ Click Here to Consult & Schedule a Scoliosis Screening via WhatsApp ]<\/strong><\/a><\/div>\n
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\ud83d\udd0d Neurotransmitters and Scoliosis: Contemporary Medical Research<\/strong><\/div>\n
Traditional biomechanical frameworks often attribute scoliosis strictly to osseous development or surface muscular imbalances. However, contemporary international clinical trials suggest that biochemical axis imbalances involving central neurotransmitters and melatonin synthesis<\/strong> are significant underlying triggers for the progression of Adolescent Idiopathic Scoliosis (AIS):<\/div>\n
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  1. Serotonin (5-HT) \u2014 Dynamic Postural Control and Bone Remodeling<\/strong>
    Serotonin functions as a vital central modulator responsible for dynamic postural control<\/strong> and baseline paraspinal muscle tone. Recent clinical data reveals that individuals with idiopathic scoliosis frequently display significantly lower blood serotonin levels compared to control groups. This central deficiency weakens the brain's symmetric neurological command over deep stabilizing paraspinal muscles and disrupts healthy bone remodeling, leaving developing vertebrae vulnerable to asymmetric rotational shifts.<\/span><\/li>\n
  2. Melatonin \u2014 Receptor Dysfunction and Asymmetric Growth Signaling<\/strong>
    Because serotonin is the biological precursor for melatonin, central serotonin deficiencies directly trigger disruptions in melatonin production and circadian regulation. Current medical focus targets signaling impairments within melatonin receptors (specifically MT2 receptors)<\/strong>. Research indicates that osteoblasts (bone-building cells) in AIS patients possess a genetic signaling defect that prevents them from utilizing melatonin properly to balance structural bone growth on both sides of the spinal axis.<\/span><\/li>\n
  3. Glycine \u2014 Inhibitory Balance of Spinal Motor Neurons<\/strong>
    Glycine is the primary inhibitory neurotransmitter within the spinal cord, responsible for regulating the excitability of motor neurons. Contemporary neurochemical studies suggest that if localized glycine receptor pathways are impaired or metabolically altered, the spinal reflex arc becomes hyper-excitable. This triggers asymmetric, chronic micro-spasms and tone imbalances<\/strong> in deep spinal muscles like the multifidus. This subconscious, neurogenic pulling is a powerful hidden driver accelerating the Cobb\u2019s angle during growth spurts.<\/span><\/li>\n<\/ol>\n<\/div>\n
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    \ud83c\udfaf The 5 Primary Classifications of Scoliosis<\/strong><\/div>\n
    To guide proper chiropractic management, scoliosis is clinically categorized into distinct types based on its developmental origins:<\/div>\n